Homotoxicology
 
Arthritis - Glucosamine supplementation
Agitation in Children
Zeel research
Preliminary Research on Euphorbium and Traumeel
Inflammation - Regulation rigidity
 
Arthritis - Glucosamine supplementation


TopTop
Agitation in Children


TopTop
Zeel research


TopTop
Preliminary Research on Euphorbium and Traumeel


TopTop
Inflammation - Regulation rigidity



Inflammation that resists initial treatment and continues is called Regulation ridgidity.
Homotoxicology differentiates between two types of regulation rigidity. The first is characterised mainly by chronic inflammation, the second by deposits of waste products and toxins.


Regulation rigidity type I

If the self-regulation does not function properly after the shock-phase, this is due to a decreased production of Th3. This could be caused, despite the fact that Traumeel“S might have been applied correctly, by an obstruction of the matrix. Th3-cells feel more resistance on their way to the lymphatic glands. They will be cloned more slowly.


The patient “hangs” in a chronic stage of inflammation.

By simultaneous application of Lymphomyosot® and Traumeel S the lymph will be better canalised and toxins will be eliminated more easily. Due to the increased lymph transport, the Th3-cells made under influence of Traumeel“S will arrive faster at the lymphatic glands in order to be cloned. The defence by the cloned Th3-cells can begin with the elimination of the Th1 and Th2-cells due to the release of TGF-b.


Regulation rigidity type II

A second type of regulation rigidity describes the opposite. The patient has been intoxicated by homotoxins, but the defence system does not react. There is hardly any phagocytosis and not many inflammation promoting factors. The patient does not show any inflammation. For many years, he has had vague complaints, (unclear deposition or impregnation phase), but there are no clinical signs.

We will have to try to induce an inflammatory reaction (force the body to vicariate towards the inflammation phase). Detox-Kit® is the ideal preparation. It activates the mesenchymal defence system that will turn against the homotoxins and bring forward the inflammation.

Once the inflammation has started (probably in the loco minoris resistentiae) we will treat the patient with a mixture of Traumeel S and Lymphomyosot®. That way we avoid the problem of the patient evolving from type II rigidity to type I rigidity.

TopTop